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MK-677

Ibutamoren · Oral GH Secretagogue · Non-Peptide

A non-peptide, orally active growth hormone secretagogue that mimics ghrelin at the GHS-R1a receptor. Technically not a peptide (it's a small molecule), but included here because it targets the same ghrelin receptor as peptide GH secretagogues and is commonly discussed alongside them. Produces sustained 24-hour GH and IGF-1 elevation from a single oral dose.

Non-peptide small molecule
Oral bioavailable
24h IGF-1 elevation
GHS-R1a agonist
Not FDA-approved
Educational content only. Not medical advice. This peptide may not be FDA-approved. Full disclaimer →
Category
GH Secretagogue (non-peptide)
Route
Oral (tablet/liquid)
Half-life
~4-6 hours (effects last 24h)
Dose
10-25 mg/day oral
Evidence
Phase II clinical

What Is MK-677?

MK-677 (ibutamoren mesylate) is a non-peptide, orally active growth hormone secretagogue developed by Merck. While it is not technically a peptide, it is universally discussed alongside peptide GH secretagogues because it targets the same ghrelin receptor (GHS-R1a) and produces similar effects.

MK-677's key advantage over peptide GHRPs is its oral bioavailability — patients take a pill instead of injecting. A single 25mg oral dose produces sustained GH pulsatility and IGF-1 elevation for a full 24 hours, making it the most convenient GH-optimizing compound available.

Core Concept
MK-677 is a non-peptide ghrelin mimetic that activates GHS-R1a with sustained kinetics due to its oral absorption and 4-6 hour half-life. Unlike injected GHRPs (which produce a single GH pulse), MK-677 maintains elevated GH pulsatility throughout the day. It increases IGF-1 by 40-90% in most studies, with effects sustained for months without the desensitization seen with hexarelin.

Structure & Sequence

MK-677
(non-peptide small molecule — no amino acid sequence)
MW: 528.7 Da (free base) · N/A (small molecule) residues
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Mechanism of Action

MK-677 activates the same GHS-R1a receptor as peptide GHRPs but with distinct pharmacokinetics. Its oral bioavailability and sustained receptor occupancy produce continuous GH pulse amplification rather than the single bolus effect of injected peptides. This more closely mimics the physiological GH secretion pattern of a younger individual.

MK-677 Oral GH Secretion
Oral dose
absorbed from GI tract
Binds
GHS-R1a (ghrelin receptor)
Sustained
GH pulsatility for 24h
Increases
IGF-1 by 40-90%
Appetite
moderate increase
Result
Sustained anabolic environment

Key Mechanisms

PathwayEffectSignificance
Oral GHS-R1a agonismSustained receptor activation from oral dosing24-hour GH pulsatility from single daily dose
IGF-1 elevation40-90% increase in IGF-1 maintained for monthsAnabolic, body composition, and recovery effects
No desensitizationUnlike hexarelin, IGF-1 elevation maintained for 12+ monthsSuitable for long-term use
Appetite increaseModerate ghrelin-mimetic hungerLess intense than GHRP-6 but more than ipamorelin
Sleep improvementIncreases duration of stage 3/4 (deep) NREM sleepGH is primarily released during slow-wave sleep

Evidence Base

StudyDesignFindingsLevel
IGF-1 elevationPhase II, n=187, 2 years25mg/day increased IGF-1 by ~40% sustained over 2 years with no tachyphylaxisLevel II
Body compositionClinical studiesIncreased lean mass (~1.8 kg) and trend toward decreased fat mass over 8 weeksLevel II
Bone densityPhase II, elderlyIncreased bone mineral density in postmenopausal women after 18 monthsLevel II
Sleep qualityClinical studiesIncreased REM and stage 4 sleep duration by ~50%Level II
SarcopeniaPhase II, elderlyImproved physical function and lean mass in elderly hip fracture patientsLevel II

Safety & Side Effects

Appetite increase: Moderate hunger increase, especially in the first 2-4 weeks. Usually diminishes.

Water retention: Edema, especially in hands and feet. GH-mediated. Usually mild.

Insulin resistance: MK-677 can worsen insulin sensitivity. Fasting glucose and HbA1c should be monitored.

Lethargy: Some users report daytime drowsiness, particularly at higher doses.

Long-term cancer risk: Theoretical concern with chronic GH/IGF-1 elevation. No increased cancer incidence in 2-year studies, but longer-term data is lacking.

Regulatory Status

JurisdictionStatus
FDANot approved. Merck discontinued development after Phase II.
WADABanned under S2 (peptide hormones and growth factors — despite being non-peptide, it targets the same pathway)
AvailabilityWidely available as a research chemical. One of the most popular GH-related compounds in biohacking communities.

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