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PT-141

Bremelanotide · Vyleesi · Melanocortin Agonist · 7 Amino Acids

A 7-amino-acid cyclic melanocortin peptide and the first FDA-approved treatment for hypoactive sexual desire disorder (HSDD) in premenopausal women. Acts centrally in the brain on MC4 receptors rather than peripherally like PDE5 inhibitors (Viagra).

7 amino acids
Cyclic peptide
FDA approved (Vyleesi)
MC4R agonist
Central mechanism
By PeptideBond Editorial Team·Sources: PubMed, FDA.gov, published clinical trials·Last updated: March 2026
Educational only — not medical advice.Disclaimer
Category
Sexual dysfunction
Route
SC injection (autoinjector)
Onset
~45 minutes
Approval
FDA 2019 (Vyleesi)
Evidence
Phase III RCTs

What Is PT-141?

PT-141 (Bremelanotide) is a cyclic 7-amino-acid peptide that activates melanocortin-4 receptors (MC4R) in the brain. Marketed as Vyleesi, it is the first and only FDA-approved treatment for hypoactive sexual desire disorder (HSDD) in premenopausal women.

PT-141 was originally developed from the tanning peptide melanotan II (which activates multiple melanocortin receptors). During clinical trials for melanotan II, researchers noticed an unexpected side effect — increased sexual arousal. This led to the development of PT-141 as a more selective MC4R agonist targeting sexual desire specifically.

Core Concept
PT-141 works through an entirely different mechanism than Viagra/Cialis (which are PDE5 inhibitors acting on blood flow). PT-141 acts in the central nervous system, binding to melanocortin-4 receptors in the hypothalamus and limbic system to modulate dopamine and oxytocin pathways involved in sexual desire and arousal. It addresses the 'wanting' (desire) rather than the 'plumbing' (blood flow).

PT-141 (bremelanotide, marketed as Vyleesi) is the only FDA-approved peptide drug specifically indicated for sexual dysfunction. Approved in June 2019 for hypoactive sexual desire disorder (HSDD) in premenopausal women, it works through a completely different mechanism than PDE5 inhibitors like sildenafil (Viagra) — it acts centrally in the brain rather than peripherally on blood flow.

PT-141 was derived from Melanotan II, a synthetic melanocortin peptide originally developed for tanning. During clinical trials, researchers noticed that Melanotan II produced unexpected sexual arousal as a side effect. PT-141 was developed as a refined version that retained the sexual effects while reducing the tanning and nausea side effects. It is a cyclic heptapeptide that activates melanocortin-4 receptors (MC4R) in the hypothalamus, modulating neural pathways involved in sexual desire and arousal.

Despite being FDA-approved, Vyleesi has had limited commercial success, partly due to its injection-based administration (subcutaneous auto-injector), the requirement to use it at least 45 minutes before anticipated sexual activity, and nausea as a common side effect. It remains the only FDA-approved on-demand treatment for female HSDD.

>Structure & Sequence

PT-141
AcNleDPheRWK
MW: 1,025.2 Da · 7 (cyclic, includes non-standard Nle and D-Phe) residues
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Mechanism of Action

PT-141 binds to the melanocortin-4 receptor (MC4R), a Gs-coupled GPCR expressed in the hypothalamus (paraventricular nucleus), amygdala, and other limbic structures involved in sexual behavior. MC4R activation increases dopaminergic signaling in the mesolimbic reward pathway, modulating the neural circuits that generate sexual desire. This central mechanism is why PT-141 works regardless of the physical cause of low desire — it targets the brain's desire circuits directly.

PT-141 Sexual Desire Pathway
SC injection
PT-141 enters bloodstream
Crosses
Blood-brain barrier
Binds
MC4R in hypothalamus
Modulates
Dopamine + oxytocin pathways
Result
Increased sexual desire

Key Mechanisms

PathwayEffectSignificance
MC4R activationAgonist binding in hypothalamic nucleiStimulates neural pathways underlying sexual desire and arousal
Dopamine modulationEnhances dopaminergic signaling in reward circuitsIncreases motivation and desire components of sexual behavior
Oxytocin releaseMC4R activation promotes oxytocin releaseMay contribute to bonding and arousal aspects
Melanocortin effectsSome residual MC1R activityCan cause transient skin darkening and facial flushing

Evidence Base

StudyDesignFindingsLevel
RECONNECT (Phase III)RCT, n=1,247 premenopausal women with HSDDStatistically significant increase in desire and decrease in distress vs placebo over 24 weeksLevel I
Male EDPhase II trialsSome efficacy in erectile dysfunction, but development focused on female HSDDLevel II
Duration of effectClinical dataSingle SC injection provides effect for ~24 hours; not a daily medicationLevel I-II

Safety & Side Effects

Nausea: The most common adverse effect, occurring in approximately 40% of patients. Nausea is typically mild-to-moderate and transient (resolving within hours), but it is significant enough to limit patient acceptance. The FDA label recommends no more than one dose per 24 hours and no more than 8 doses per month.

Blood pressure: Transient blood pressure increases (average 2-3 mmHg systolic) have been observed after injection. Vyleesi is not recommended for patients with uncontrolled hypertension or cardiovascular disease.

Skin hyperpigmentation: Due to PT-141's melanocortin activity, darkening of the skin (particularly gums, face, and breasts) has been reported with repeated use. The FDA label specifically warns about this effect, which may be persistent.

Focal hyperpigmentation: PT-141 should not be used in patients with a history of skin conditions sensitive to melanocyte activation, as it could theoretically affect nevi (moles) or other pigmented lesions.

Regulatory Status

JurisdictionStatus
FDAApproved: Vyleesi (bremelanotide) for HSDD in premenopausal women (2019). SC autoinjector.
EMANot approved in EU
WADANot specifically banned

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