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Tesamorelin

Egrifta · GHRH Analog · HIV Lipodystrophy

A 44-amino-acid modified GHRH analog with a trans-3-hexenoic acid group attached to the N-terminal tyrosine. The only GHRH-based drug currently FDA-approved for therapeutic use in the US — specifically for reducing excess visceral abdominal fat in HIV-infected patients with lipodystrophy.

44 amino acids
Modified GHRH analog
FDA approved (Egrifta)
HIV lipodystrophy
Visceral fat reduction
By PeptideBond Editorial Team·Sources: PubMed, FDA.gov, published clinical trials·Last updated: March 2026
Educational only — not medical advice.Disclaimer
Category
GH Releasing / Fat reduction
Route
SC injection (daily)
Dose
2 mg/day
Approval
FDA 2010 (Egrifta)
Evidence
Phase III RCTs

What Is Tesamorelin?

Tesamorelin is a synthetic 44-amino-acid analog of human GHRH with a single modification: a trans-3-hexenoic acid group conjugated to the N-terminal tyrosine residue. This modification improves stability and bioavailability compared to native GHRH.

It is marketed as Egrifta and is FDA-approved specifically for reducing excess visceral abdominal fat (lipodystrophy) in HIV-infected patients on antiretroviral therapy. It is the only GHRH-based drug currently approved for therapeutic use in the United States.

Core Concept
Tesamorelin works by the same GHRH receptor mechanism as sermorelin and CJC-1295 — binding to pituitary GHRH receptors and stimulating endogenous GH production. The GH released then promotes lipolysis (fat breakdown) specifically in visceral adipose tissue. Unlike direct GH injection, tesamorelin preserves the pulsatile pattern and allows negative feedback to prevent supraphysiological GH levels.

Tesamorelin (marketed as Egrifta and Egrifta SV) is the only FDA-approved GHRH analog for therapeutic use in the United States. It was approved in 2010 specifically for the reduction of excess abdominal fat (lipodystrophy) in HIV-infected adults on antiretroviral therapy — a condition where metabolic disruption from both the virus and its treatment causes abnormal fat accumulation in the trunk region.

Tesamorelin is a modified form of GHRH(1-44) with a trans-3-hexenoic acid modification at the N-terminus that enhances receptor binding and extends duration of action compared to native GHRH. Unlike direct GH replacement, tesamorelin works through the body's own pituitary GH production, maintaining physiological feedback regulation.

Beyond its approved indication, tesamorelin has generated significant interest in anti-aging medicine and in the treatment of NAFLD/MASLD (metabolic dysfunction-associated steatotic liver disease). A 2019 study published in The Lancet HIV showed that tesamorelin reduced liver fat by 37% in HIV-infected patients with NAFLD — a finding that has implications well beyond the HIV population.

>Structure & Sequence

Tesamorelin
hexenoic-YADAIFTNSYRKVLGQLSARKLLQDIMSRQQGESNQERGARARL
MW: 5,135.9 Da · 44 + N-terminal modification residues
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Mechanism of Action

Tesamorelin activates the GHRH receptor on pituitary somatotrophs, stimulating GH production through the Gs-cAMP-PKA signaling cascade. The resulting GH increase preferentially mobilizes visceral fat through activation of hormone-sensitive lipase in visceral adipocytes, which have higher GH receptor density than subcutaneous fat.

Tesamorelin Mechanism
Binds
GHRH receptor
Stimulates
Endogenous GH release
GH promotes
Visceral fat lipolysis
Reduces
Trunk fat (measured by CT)
Result
Improved body composition

Key Mechanisms

PathwayEffectSignificance
GHRH-R activationFull agonist at pituitary GHRH receptorStimulates endogenous GH production preserving pulsatility
Visceral fat reductionGH-mediated lipolysis in visceral adipose17% reduction in trunk fat in Phase III trials
IGF-1 increaseGH-driven hepatic IGF-1 productionIncreases IGF-1 within normal physiological range
Liver fat reductionReduces hepatic steatosisPotential benefit for NAFLD/NASH (being studied)
No glucose worseningUnlike direct GH, minimal impact on insulin sensitivity at approved doseSafer metabolic profile for diabetic patients

Evidence Base

StudyDesignFindingsLevel
Phase III (HIV lipodystrophy)RCT, n=81617% reduction in visceral adipose tissue at 26 weeks vs placebo. FDA approval basis.Level I
NAFLD/NASHPhase II, n=61Reduced hepatic fat fraction by 37% in HIV patients with fatty liverLevel II
Cognitive functionClinical studyImproved cognitive function in HIV patients, possibly via IGF-1 effects on hippocampusLevel II-III
Long-term safetyExtension studies, 2+ yearsSustained fat reduction; no significant safety concerns. IGF-1 remained in normal range.Level I-II

Safety & Side Effects

FDA-approved safety profile: As an FDA-approved drug, tesamorelin has well-characterized safety from clinical trials and post-marketing surveillance. The most common side effects are injection site reactions (redness, swelling, pain) occurring in approximately 10% of patients.

Arthralgia: Joint pain is reported in approximately 10-13% of patients and is likely related to increased GH/IGF-1 levels. The effect is typically mild and dose-dependent.

Peripheral edema: Fluid retention and swelling, consistent with GH elevation, occurs in a subset of patients.

Glucose effects: Tesamorelin may slightly impair glucose tolerance, consistent with GH's counter-regulatory effects on insulin. Glucose monitoring is recommended, particularly in patients with pre-diabetes or diabetes.

Hypersensitivity: Rare hypersensitivity reactions including rash, urticaria, and pruritus have been reported. Tesamorelin should be discontinued if hypersensitivity occurs.

Pregnancy: Tesamorelin is contraindicated in pregnancy due to potential effects on fetal development. It should be discontinued upon pregnancy confirmation.

Regulatory Status

JurisdictionStatus
FDAApproved: Egrifta (tesamorelin) for HIV lipodystrophy (2010). Daily SC injection.
Off-labelIncreasingly used off-label for general visceral fat reduction and anti-aging
WADABanned under S2

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