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Tesamorelin

Egrifta · GHRH Analog · HIV Lipodystrophy

A 44-amino-acid modified GHRH analog with a trans-3-hexenoic acid group attached to the N-terminal tyrosine. The only GHRH-based drug currently FDA-approved for therapeutic use in the US — specifically for reducing excess visceral abdominal fat in HIV-infected patients with lipodystrophy.

44 amino acids
Modified GHRH analog
FDA approved (Egrifta)
HIV lipodystrophy
Visceral fat reduction
Educational content only. Not medical advice. This peptide may not be FDA-approved. Full disclaimer →
Category
GH Releasing / Fat reduction
Route
SC injection (daily)
Dose
2 mg/day
Approval
FDA 2010 (Egrifta)
Evidence
Phase III RCTs

What Is Tesamorelin?

Tesamorelin is a synthetic 44-amino-acid analog of human GHRH with a single modification: a trans-3-hexenoic acid group conjugated to the N-terminal tyrosine residue. This modification improves stability and bioavailability compared to native GHRH.

It is marketed as Egrifta and is FDA-approved specifically for reducing excess visceral abdominal fat (lipodystrophy) in HIV-infected patients on antiretroviral therapy. It is the only GHRH-based drug currently approved for therapeutic use in the United States.

Core Concept
Tesamorelin works by the same GHRH receptor mechanism as sermorelin and CJC-1295 — binding to pituitary GHRH receptors and stimulating endogenous GH production. The GH released then promotes lipolysis (fat breakdown) specifically in visceral adipose tissue. Unlike direct GH injection, tesamorelin preserves the pulsatile pattern and allows negative feedback to prevent supraphysiological GH levels.

Structure & Sequence

Tesamorelin
hexenoic-YADAIFTNSYRKVLGQLSARKLLQDIMSRQQGESNQERGARARL
MW: 5,135.9 Da · 44 + N-terminal modification residues
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Mechanism of Action

Tesamorelin activates the GHRH receptor on pituitary somatotrophs, stimulating GH production through the Gs-cAMP-PKA signaling cascade. The resulting GH increase preferentially mobilizes visceral fat through activation of hormone-sensitive lipase in visceral adipocytes, which have higher GH receptor density than subcutaneous fat.

Tesamorelin Mechanism
Binds
GHRH receptor
Stimulates
Endogenous GH release
GH promotes
Visceral fat lipolysis
Reduces
Trunk fat (measured by CT)
Result
Improved body composition

Key Mechanisms

PathwayEffectSignificance
GHRH-R activationFull agonist at pituitary GHRH receptorStimulates endogenous GH production preserving pulsatility
Visceral fat reductionGH-mediated lipolysis in visceral adipose17% reduction in trunk fat in Phase III trials
IGF-1 increaseGH-driven hepatic IGF-1 productionIncreases IGF-1 within normal physiological range
Liver fat reductionReduces hepatic steatosisPotential benefit for NAFLD/NASH (being studied)
No glucose worseningUnlike direct GH, minimal impact on insulin sensitivity at approved doseSafer metabolic profile for diabetic patients

Evidence Base

StudyDesignFindingsLevel
Phase III (HIV lipodystrophy)RCT, n=81617% reduction in visceral adipose tissue at 26 weeks vs placebo. FDA approval basis.Level I
NAFLD/NASHPhase II, n=61Reduced hepatic fat fraction by 37% in HIV patients with fatty liverLevel II
Cognitive functionClinical studyImproved cognitive function in HIV patients, possibly via IGF-1 effects on hippocampusLevel II-III
Long-term safetyExtension studies, 2+ yearsSustained fat reduction; no significant safety concerns. IGF-1 remained in normal range.Level I-II

Safety & Side Effects

Injection site reactions: Erythema, pruritus, and pain at injection site are the most common side effects (~25%).

Arthralgia: Joint pain reported in ~13% of patients. Related to GH/IGF-1 increase.

Fluid retention: Peripheral edema and carpal tunnel symptoms can occur.

Theoretical cancer risk: GH/IGF-1 elevation could theoretically promote tumor growth. FDA label warns about this. Monitor for malignancy.

Regulatory Status

JurisdictionStatus
FDAApproved: Egrifta (tesamorelin) for HIV lipodystrophy (2010). Daily SC injection.
Off-labelIncreasingly used off-label for general visceral fat reduction and anti-aging
WADABanned under S2

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